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Drug Rehab Hawaii is here to help people with drug and/or alcohol abuse problems in Hawaii. find treatment options. Due to our diverse networking system we can find a treatment option tailored to each individuals specific situation and needs. We are able to provide all phases of recovery included but not limited to, alcohol and/or drug intervention, drug and/or alcohol detox, in-patient treatment, out-patient treatment, short term treatment (30 days or less), long term treatment (90 days or longer).

We design personalized treatment programs to provide each abuser with the greatest chance of a successful recovery outcome. Our comprehensive networking system works hand in hand with all of the drug treatment centers in Hawaii. At Drug Rehab Hawaii we know that each individual is unique and are treated as such. Deciding upon a treatment option in Hawaii, or anywhere can be a daunting task for any individual or family, we will guide you through each step of a comprehensive treatment plan for you or your loved one. We are determined in our mission, that every drug and/or alcohol abuser in Hawaii. that has a desire to change their life will be given a chance to recover from their addiction and we are dedicated to ensuring that they are given the opportunity to do so. We realize that each individual in Hawaii. is in a different financial situation and we will find treatment options for each individual regardless of their financial situation. No matter what your financial situation everyone will receive the treatment help they are looking for.          866-407-4380

OxyContin Side Effects

OxyContin, approved by the FDA in 1995, an opium derivative, which is the same active ingredient in Percodan and Percocet. OxyContin is intended for use by terminal cancer patients and chronic pain sufferers. It has been linked to at least 120 overdose deaths nationwide.

Respiratory depression is the chief hazard from all opioid usage. Respiratory depression occurs most frequently in elderly or debilitated patients, usually following large initial doses in non-tolerant patients, or when opioids are given in conjunction with other agents that depress respiration. Common opioid side effects are constipation, nausea, sedation, dizziness, vomiting, headache, dry mouth, sweating, and weakness.

Oxycodone should be used with extreme caution in patients with significant chronic obstructive pulmonary disease and in patients having a substantially decreased respiratory reserve, hypoxia, hypercapnia, or preexisting respiratory depression. In such patients, even usual therapeutic doses of oxycodone may decrease respiratory drive to the point of apnea. In these patients alternative non-opioid analgesics should be considered, and opioids should be employed only under careful medical supervision at the lowest effective dose. Oxycodone causes miosis, even in total darkness. Pinpoint pupils are a sign of opioid overdose but are not pathognomonic. Marked mydriasisrather than miosis may be seen due to hypoxia in overdose situations.

• Gastrointestinal Tract and Other Smooth Muscle
  Oxycodone causes a reduction in motility associated with an increase in smooth muscle tone in the antrum of the stomach and duodenum. Digestion of food in the small intestine is delayed and propulsive contractions are decreased. Propulsive peristaltic waves in the colon are decreased, while tone may be increased to the point of spasm resulting in constipation. Other opioid-induced effects may include a reduction in gastric, biliary and pancreatic secretions, spasm of sphincter of Oddi, and transient elevations in serum amylase.
• Cardiovascular System
  Oxycodone may produce release of histamine with or without associated peripheral vasodilation. Manifestations of histamine release and/or peripheral vasodilation may include pruritus, flushing, red eyes, sweating, and/or orthostatic hypotension.
• Concentration--Efficacy Relationships (Pharmacodynamics)
  Studies in normal volunteers and patients reveal predictable relationships between oxycodone dosage and plasma oxycodone concentrations, as well as between concentration and certain expected opioid effects. In normal volunteers these include pupillary constriction, sedation and overall "drug effect" and in patients, analgesia and feelings of "relaxation." In non-tolerant patients, analgesia is not usually seen at a plasma oxycodone concentration of less than 5&10 ng/mL.

As with all opioids, the minimum effective plasma concentration for analgesia will vary widely among patients, especially among patients who have been previously treated with potent agonist opioids. As a result, patients need to be treated with individualized titration of dosage to the desired effect. The minimum effective analgesic concentration of oxycodone for any individual patient may increase with repeated dosing due to an increase in pain and/or the development of tolerance.

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